By C. Meyers
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49 Lim and colleagues65 reviewed 363 individuals with ARL (including DLBCL and BL) from 1982 to 2003, including 262 in the pre-HAART era, and 101 in the HAART era. 2 months in DLBCL). 65 Cortes and colleagues used HyperCVAD (hyperfractionated cyclophosphamide, vincristine, doxorubicin, and dexamethasone alternating with high-dose methotrexate and cytarabine) regimen in 13 patients with HIV-related BL, nine of whom also received HAART. 66 While Wang and colleagues, in a retrospective study, compared 14 HIV-infected individuals with BL with 24 non-HIV-infected individuals with BL who received CODOX-M/IVAC (cyclophosphamide, doxorubicin, high-dose methotrexate/ifosfamide, etoposide, and high-dose cytarabine) or less intensive regimens.
185 In the pre-HAART era, the AIDS Clinical Trials Group (ACTG) developed a staging system based on tumor extent (T), CD4 cell count (L), and presence of systemic illness (S). Two different risk categories were noted based on this staging system. A good risk is defined as (T0L0S0) and is seen in disease involving the skin, and/or presence of minimal oral disease, CD4 > 150 µL, no opportunistic infectious (OI)/B symptoms and performance status (PS) > 70. 186 concluded from epidemiological, clinical, staging, and survival data from 211 patients in two Italian prospective cohort studies that in the era of HAART, a refinement of the ACTG staging system is needed.
168 Adverse side effects from interferon-alpha encompass fever, chills, neutropenia, hepatotoxicity, and cognitive impairment. Anti-HHV-8 Therapy The discovery of HHV-8 in all forms of KS has raised the possibility of using antiviral agents to target this virus. HIV-infected individuals that used foscarnet instead of ganciclovir to treat cytomegalovirus disease had a considerably longer 2. 205 A pilot study using cidofovir for the treatment of AIDS-associated and classic KS showed no effect on disease progression, no decrease in viral load of HHV-8 among seven patients, and no change in expression of early lytic and latent gene expressions from cutaneous KS lesions.